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Specifically, as proposed in a hypothetical model shown in Figure 7(b), if a protein or a small molecule target could be designed under diabetic conditions to reduce metabolic pressure on complex I, that is, relaying excess electrons from NADH to CoQ, such a target could serve as a potential therapeutic approach by restoring NADH/NAD + redox balance in the absence of enhanced proton pumping and ROS production

During the preparation of this work, the author(s) used ChatGPT to correct the grammar, language and used Google Gemini to create Figure 1

Epigenetic silencing of microRNA-137 enhances ASCT2 expression and tumor glutamine metabolism

In other words, if youre not recycling glutathione well youre at more risk of developing autoimmune disease or flaring an existing thyroid autoimmunity