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The mechanisms most relevant to knee pain: VEGF upregulation stimulates new blood vessel formation in cartilage and connective tissue, areas that are notoriously slow to heal due to limited blood supply Nitric oxide modulation regulates inflammation and promotes vasodilation, improving blood flow to damaged tissue Collagen synthesis accelerates organized collagen deposition at repair sites instead of disorganized scar tissue Tendon-to-bone healing particularly relevant at the patella tendon and other knee attachment points Animal studies across rat, rabbit, and canine models showed consistent findings across these pathways

Through its multi-target mechanism of action (anti-inflammation, mucosal repair, angiogenesis), it provides a new option for patients who are ineffective or intolerant to traditional treatments

UVB: 312 nm, 0.91 mW/cm 2 , 0.08 J/cm 2 ) in human epidermal keratinocytes (ScienCell) 178

Current guidelines in treatment recommend preserving and maintaining the structural integrity of the meniscus whenever possible