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Pathological outcomes are more likely to emerge when adaptive mechanisms are overwhelmed by chronic stressors, persistent chronodisruption, or underlying disease processes

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The in vitro cell-based mechanistic studies have reported that PNS can upregulate the angiogenic capacity of endothelial progenitor cells by activating the Wnt/-catenin signaling pathway and synergistically regulate the HIF1-/VEGFA/VEGFR2 signaling axis to promote angiogenesis, thereby improving cerebral microcirculation disorders (Zhu et al., 2021

Consequently, this may result in IGF-1 LR3 being more potent but with a shorter duration of action compared to recombinant Insulin-like Growth Factor 1 (rhIGF-1)