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Recent evidence suggests that lung resident mesenchymal stem cells (LR-MSCs) are precursors of myofibroblasts and also can be induced to differentiate into many other cell types that may participate in lung repair or contribute to the development of pulmonary diseases [6, 7]

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Interestingly, mannose binding lectin (MBL), a lectin involved in the recognition of infectious agents by the innate immune system, is significantly down-regulated in PD patients

This variability exists on multiple interconnected levels, demanding a far more significant approach than one size that fits all strategy