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*Correspondence: Weina Li, [email protected] Meng Li, [email protected] These authors have contributed equally to this work This article was submitted to Drug Metabolism and Transport, a section of the journal Frontiers in Pharmacology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Rat amylin analogs with these affinity-enhancing mutations also significantly enhanced potency for calcitonin receptor activation by 5- to 11-fold compared to endogenous rat amylin and a clinical drug pramlintide (Figure 9D and Table 3)

Behav Neurol 2025:9627699

Jang and Kim, 2020)