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Our results revealed that the obtained liposomal drug depot exhibited regular morphology and uniform size, typically ranging between 200 and 300 nm, which facilitated their penetration into the inflammatory microenvironment through the extravasation through leaky vasculature and subsequent inflammatory cell-mediated sequestration (ELVIS) effect [38]

For instance, decreased NAD availability can lead to compromised mitochondrial function, resulting in increased oxidative stress and neuronal damage