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Following TMZ treatment, these alterations were reversed through the downregulation of TfR and ACSL4, as well as the upregulation of FTH and GPX4, leading to increased cell survival rates

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Figure 1 6.6 Therapeutic and translational opportunities targeting mitochondrial dysfunction in infection Targeting mitochondrial dysfunction offers a promising strategy for HDTs

Table 3 summarizes the most commonly used treatments to reduce IRI, which include adenosine agonists, endothelin antagonists, antioxidants, complement activation inhibitors, apoptosis inhibitors, anti-inflammatory and proangiogenic, metabolic inhibitors, bioactive gases, traditional Chinese medicine, cell-based therapy, etc