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In light of the absence of direct treatment recommendations for reducing oxidative stress, there is a continuing need for fundamental research that utilizes innovative therapeutic approaches

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The emerging evidence that eosinophils are pathogenic cells is prompting the development of additional eosinophil-directed therapeutics such as agents that interfere with the eosinophil-specific surface receptors sialic acid-binding Ig-like lectin 8 (SIGLEC-8) and the epidermal growth factor-like module containing mucin-like hormone receptor 1 (EMR1)

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