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In addition, SREBP1c indirectly contributes to the development of hepatic insulin resistance, since enhanced lipogenesis and subsequent accumulation of harmful lipid species, such as diacylglycerides, may interfere with insulin signaling (as discussed below)

In fact, both pharmacological alteration of the mitochondrial potential (Hacker and Medler, 2008) and blocking of NCX, induced Ca 2+ signals in taste bud cells (Laskowski and Medler, 2009)