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Description

In most cases, no complicated preparation is required

65 Mice homozygous for the physiologically important AKT substrate TBC1D4 Thr 649 knock-in exhibit impaired insulin-stimulated myocellular GLUT4 translocation and induction of glucose intolerance

Data availability All data supporting the findings of this study are available within the paper and its Supplementary Information as well as the Source Data file