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In serous ovarian cancer (SOC), ALOX5AP is highly expressed and is associated with poor prognosis and aggressive disease features, such as lymphatic invasion, resistance to platinum chemotherapy, and poor survival rates

KDM5B knockdown in breast cancer cells activated the AMPK protein, reduced the level of lipid metabolism, and inhibited the epithelial-mesenchymal transition (EMT), and reduced their growth and migration ability, revealing novel theoretical evidence for the clinical regulation of tumor metabolism by targeting related genes

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USP25 regulates KEAP1-NRF2 anti-oxidation axis and its inactivation protects acetaminophen-induced liver injury in male mice