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demonstrated that MPH promotes ROS generation via activation of RAC1-dependent NADPH oxidase (NOX) and c-Src activation [149]

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In relative to the -cell of the sham group, LOsG resulted in 3.3-fold increase of nuclear TXNIP (Fig

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The pharmacologic profile of isoproterenol, a synthetic, non-selective -adrenergic agonist

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ROS inhibit endothelial nitric oxide synthase (eNOS), reducing NO production and compromising vasodilation

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