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An essential role for phoP in Mycobacterium tuberculosis virulence

At the same time, oxidative stress, which is caused by mitochondrial damage, activation of NADPH oxidase, and other environmental or treatment-related insults, promotes tumourigenesis through a cascade of events including: oxidative DNA adducts (8-oxo-dG), lipid peroxidation (4-HNE), protein oxidation, redox-regulated oncogene activation and mutagenesis, inhibition of the DNA repair machinery, and a vicious cycle of genomic instability accompanied by inflammatory signalling (178180)

Carnitine increases the use of fat as an energy source

Essential strategies to manage oxidative stress include: Reduce Toxic Burden : Environmental exposure to toxins, heavy metals, pollutants, and industrial chemicals can trigger oxidative stress by catalyzing ROS production, promoting free radical production, depleting antioxidants, and inhibiting their protective functions