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Other causes of liver injury were excluded, including HBV, HCV, and HIV infections, drug-induced hepatitis, autoimmune liver disease, fatty liver disease, malignant tumors, and pregnant women

Pierce Memorial Bench 3

The results suggested that TB-500 not only potentially enhanced the viability, angiogenesis, and migratory ability of HUVEC but possibly also promoted the expression of angiopoietin-2 (Ang2), TEK receptor tyrosine kinase 2 (tie2), vascular endothelial growth factor A (VEGFA), NOTCH1 intracellular domain (N1ICD), Notch receptor 3 (Notch3), NF-B, and phosphorylated (p)-p65 in HUVEC

McCullough et al., 2022) and four casecontrol studies (Norell et al., 1986