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Therefore, this study confirms that pioglitazone activates AMPK, which counteracts high glucose effects by stabilizing GLS1 through HSP75/TRAP1, suggesting that GLS1 stability may protect against -cell dysfunction via the GSH antioxidant system [11]

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The MEK inhibitor selumetinib has been reported as a low-toxicity and efficacious intervention in a trial for children with NF1 and inoperable pNFs