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The disease is caused by a mutation in the ATP7B gene, which codes for a protein that facilitates the incorporation of copper into proteins (such as ceruloplasmin) and also the transportation of copper into vesicles that allow it to be secreted in bile.1 The critical effect of a mutation in ATP7B is diminished copper secretion into bile, which leads to excess copper accumulation in the hepatocyte

International Journal of Biological Macromolecules, 253 , 127554

It was suggested that the differentiated production of IL-1 and IL-8 may be linked to the type of product applied, either an irritant or a sensitizer 39,40

Potential adverse effects of amphetamine treatment on brain and behavior: a review