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(2021) reported that m6A modification appeared to enhance Beclin-1-mediated autophagy and consequently trigger ferroptosis in human HSCs, which is further confirmed by silencing YT521-B homology domain family, a critical m6A reader protein for sustaining Beclin-1 mRNA stability

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Scientific evidence[14,15] of the critical role of serotonin in the pathogenesis of depression was instrumental to the development of some of the current antidepressant drugs (fluoxetine and sertraline [SERT]) that selectively inhibit the reuptake of serotonin at serotonin transporters, and thereby increase serotonin concentration within the synaptic cleft[15,16]

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