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Many of the FDA advisers and internal staff who oversaw those decisions no longer work for the agency

Conclusion and perspectives Lactylation has emerged as a multifaceted regulatory mechanism that not only modulates gene transcription and signal transduction but also profoundly influences protein characteristics and functions

The pathogenesis of myelodysplastic syndrome (MDS) and erythropoiesis involve PLC-1/protein kinase C (PKC) signal transduction, especially nuclear PLC-1 gene (94).PLC-1a is a negative regulator of erythroid differentiation, and which is reduced in erythropoietin-responder MDS patients and in normal hematopoietic stem cell progenitors induced to erythroid differentiation (95)