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This dual regulation by distinct sirtuins suggests a crosstalk or synergy between different post-translational modifications

In summary, our results suggest that AGH treatment not only restores the fiber type composition but also significantly improves muscle force impairments in the dystrophic skeletal muscle of mdx mice

Sox9 knockdown led to apoptosis of rBM-MSCs by upregulating caspase 3/7 activity and downregulating anti-apoptotic protein Bcl-2

GHK-Cu suppresses inflammation by lowering the level of acute-phase inflammatory cytokines, including TGF- and TNF-, and reduces oxidative damage by modulating iron levels [21, 22]