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Further studies have shown that genetic knockout of GPX4 induces cell death with characteristic features of ferroptosis, genetically confirming its central role in maintaining cellular lipid homeostasis and defending against oxidative damage (Dixon et al., 2012

Blood Cells, Molecules and Diseases 33 (2), 132140

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In the following sections, we will use this framework to examine emerging evidence of the impact of G6PD-d on three major ASCVD-mediating immune cells: macrophages, T cells and B cells