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For example, a large expansion of the astacin protease and CAP gene families coincided with the emergence of parasitism in the animal parasitic Strongyloides nematodes [10] whilst gene duplication is proposed to have been an important driver of parasitism in the Orobanchaceae plant parasites [11]

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Such a staged program would help determine not only whether a compound is protective, but also whether it acts by stabilizing an upstream contact-site defect, limiting secondary amplification, or merely delaying terminal bioenergetic failure

This can lead to the development of innovative treatments that address the root cause of the condition rather than just managing symptoms