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The cited pharmacological study has demonstrated that curdione modulates redox complexes by disrupting Keap1-Trx1 interactions while enhancing Trx1-GPX4 binding, thereby attenuating post-infarction ferroptosis through Keap1/Trx1/GPX4 axis regulation (64)

Once inside the brain, pyridostigmine directly enhances neuronal excitability and induces an early transcriptional response that increases c-fos and AChE mRNAs (Friedman et al, 1996)