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Therefore, transsulfuration pathway likely promotes the cardiovascular protection via redox regulation

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PMID: 65 NicotraA

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Medler, 2015), and metabolic stimuli may affect intracellular Ca 2+ homeostasis by interfering with mitochondrial activity: in type II cells, changes in mitochondrial potential induced by glucose metabolism may decrease mitochondrial Ca 2+ buffering and affect intracellular Ca 2+ transients (Hacker and Medler, 2008

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Clinicians must balance providing enough Se to correct deficiencies and prevent toxic effects [28]

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