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To dissect the biochemical derangements imposed by chronic hyperglycaemia, a three-dimensional, tissue-engineered wound model has been engineered that faithfully recapitulates the accumulation of AGEs and the attendant impairment of fibroblast proliferation, angiogenic signaling, and extracellular-matrix turnover that underlie the stalled diabetic repair program (129)

The process seems straightforward, but it meets a difficulty known as first-pass metabolism

If left untreated, these low blood sugar events could even lead to coma or death

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