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Since bacteria exhibit different modes of response to different antibiotic concentrations,[] we used moxifloxacin at MBC levels to maintain the antibiotic concentration always high, with negligible changes in its concentration during the exposure, reported earlier.[] We did not study antibiotic response at single-cell level as antibiotic-exposed single cells in isolation respond differently to antibiotics, unlike the single cells in a population, depending on the antibiotic concentration and exposure duration.[] Formation of three reactive oxygen species in the moxifloxacin-exposed Mycobacterium smegmatis cultures The genetic resisters to moxifloxacin were formed from the cells in the ASP containing the three ROS and Fe (II) at significantly elevated levels

Microglial depletion reduced CD3 + and CD8 + T cells and attenuated tau pathology in TE4 mice

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Within this complex pathophysiological framework, a wide array of inflammatory cytokines and markers are involved, each playing an indispensable role and notably impacting the developmental trajectory of CHD