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Activated XBP1 drives an abnormal triglyceride biosynthesis program, causing further lipid accumulation within tDCs, ultimately hindering their antigen-presenting ability and preventing them from effectively activating tumor-infiltrating T cells, thereby suppressing the anti-tumor immune response [159]

Elucidating the intricate network of interactions and unravelling the specific molecular events driving oxidative stress will be crucial for developing targeted therapeutic approaches and improving our understanding of diabetes pathogenesis as a whole

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It is noteworthy that the longer half-life of ONOO compared to OH may increase the likelihood of its reaction with hydrogen gas