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These metabolic shifts increase energy expenditure, suppress lipogenesis, and rejuvenate muscle stem cell function, providing a unique mechanism for combating obesity, fatty liver, and age-related muscle decline 1-3

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Insulin normally suppresses gluconeogenesis while promoting lipogenesis, however, under insulin resistant states such as T2D, hepatic lipogenesis remains insulin sensitive and suppression of gluconeogenesis is insulin resistant, leading to a metabolic state called selective hepatic insulin resistance (199)