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GPX2 deletion alters endoderm in vitro differentiation To directly assess the GPX2 role in cell differentiation, we generated a homozygous GPX2 loss-of-function model in hPSCs by deleting the entire GPX2 gene (genomic coordinates, chr14:64,939,301-64,942,926, Hg38) (Fig

Analysis of the signaling pathways that may be influenced by the combination of both compounds revealed that treatment with UroA and bortezomib increased p53 and p21 expression in both bortezomib-sensitive and -resistant lines

This confusion has grown as injectable therapies become more common nationwide