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In animal models of ALD, serum and hepatic concentrations of TNF- are significantly elevated in patients with sAH and closely correlate with the extent of liver injury [17]

In the present study, the significant reductions of liver and plasma GSH (Table 3) and cystathionine concentrations (Table 2), in addition to the significant correlation between these two compounds, suggested that modulation of the methyl balance and the consequent reduction of Hcy can down-regulate the trans-sulfuration pathway in a dose-dependent response

CSE is suggested to exhibit its anti-atherosclerotic effect through 3 mechanisms, namely the reduction of chemotactic factors inter cellular adhesion molecule-1 (ICAM-1) and CX3CR1, inhibition of macrophage lipid uptake, and induction of smooth muscle cell apoptosis via MAPK pathway [3]

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