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Selective impairment of this carrier-mediated transport can contribute to selective vulnerability of tissues to specific toxins: for example, chronically ethanol-fed hepatocytes become selectively depleted of mitochondrial glutathione because of defective carrier-mediated transport, which makes the hepatocytes susceptible to damage by reactive oxygen species generated from the oxidative metabolism of ethanol (22)

Trophoblast cells undergo various forms of RCD, which can influence their proliferation, invasion, and uterine spiral artery remodeling

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Baseline oxidative stress and glutathione biomarkers should be measured before starting supplementation, then rechecked every 812 weeks