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In addition, TMAO was also found to increase the level of ROS and upregulate the expression of TLR4 in cultured cells (Li et al., 2019b)

(1992, Biochem J) demonstrated that IGF-1 and its variants including Long R3 IGF-1 are anabolic in dexamethasone-treated rats, confirming the utility of IGF-1 LR3 in models of catabolic states and muscle wasting where native IGF-1 activity is suppressed by glucocorticoid-driven IGFBP upregulation