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Description

A key mechanism involves the deactivation of hyperactivated P38 (MAPK) and P53 targets in both kidney and liver tissues [48]

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This restoration leads to improved mitochondrial health, enhanced CCO activity, angiogenesis (via VEGF signaling), and attenuation of cardiac hypertrophy [35, 178]

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Protease-Activated Receptor 4: A Critical Participator in Inflammatory Response

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