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It is a multifactorial process driven by the accumulation of genetic mutations over time, shaped by a combination of inherited susceptibility, environmental exposures, and lifestyle factors. These interacting variables determine whether a cell maintains normal growth control or progresses into malignancy

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Research has shown that METTL3 and FTO had opposite effects on this process: FTO enhanced adipocyte differentiation, while METTL3 negatively impacted adipogenesis [111, 112]