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Pearson and McConnell [122] reported lack of information in relation to the MC concentration levels in living organisms and rate of its transport through food chains

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However, factors associated with overnutrition and insulin resistance likely play a role in the maladaptation of mitochondrial oxidative function that leads to inefficient oxidative flux, accumulation of lipotoxic intermediates and the progression from isolated steatosis to NASH (71,93)

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