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The resultant surplus of glutamate excessively stimulates NMDA receptors on spinal neurons, resulting in calcium influx, oxidative damage, and neuronal hyperexcitability [65, 117]

Nat Commun (2021) 12(1):3073

The DMN is composed of interconnected midline structures, including the sgACC, ventromedial PFC (vmPFC), dorsomedial PFC, precuneus/PCC complex, and mesotemporal structures

In summary, SLC7A5 overexpression was able to reverse the protective effects of YTHDF1 knockdown, revealing the critical role of SLC7A5 in the YTHDF1-mediated cytoprotective mechanism