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Li et al, 2011

Likewise, considering the studies (i.e., [23,79,80]) it seems [21] that early therapy with endothelin receptor antagonists may only delay rather than stop the development of pulmonary hypertension (whereas BPC 157 eliminated monocrotaline-induced pulmonary arterial hypertension)

So if something goes wrong and the GSSG accumulates, it inhibits an enzyme that relies on the correct oxidation of the ER environment to operate a protein quality control system. Moreover, they discovered, when misfolded proteins dont pass quality control, they wont get exported, so they too pile up in the ER

As a result, ROS have the potential of causing peroxidation of membrane lipids, aggression of tissue membranes and proteins, or damage to DNA and enzyme and generally by oxidizing low-density lipoproteins (LDL)