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In turn, this inhibits respiratory chain activity and mitophagy and induces oxidative damage and apoptosis in pancreatic islet -cells, leading to enhanced insulin resistance ( In vivo experiments further confirmed that PAA significantly reduced blood urea nitrogen (BUN) and proteinuria levels in STZ-induced DKD mice, inhibited the expression of IL-1 and TNF- in renal tissues, significantly increased the levels of LC3 and ATG5 proteins, and downregulated the levels of marker proteins such as p62 and FUNDC1, suggesting that PAA can improve renal dysfunction in DKD mice by downregulating FUNDC1 activation of mitophagy (Wu et al., 2023)

Additionally, the increased CHAC1 mRNA decay, GSH accumulation, ROS scavenging, drug resistant phenotypes in vitro and in vivo induced by GDIL overexpression was blocked when fragment 1 (1200 nt) in GDIL was deleted (Supplementary Fig

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