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The inclusion of HA facilitated the targeted delivery of LMMH to the tumor site following systemic circulation, resulting in the rapid release of Mn 2+ and LT by exploiting GSH in the TME

Drug resistance can be divided into acquired and intrinsic resistance [7, 8]

Similarly, the timing of intervention, whether administered before, concurrent with cyclophosphamide, may determine the extent of protection conferred, as it can affect the agents ability to precondition tissue, interrupt ongoing damage, or promote recovery

[1] [10] Another consequence of the differences in the primary structure concerns the net surface charge, which is -6 for the M subunit and +1 for the H subunit