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Meanwhile, myeloid-cell specific GPX4 haploinsufficient mice presented with SLE-like manifestations such as spontaneous skin lesions and systemic damage, further validating that ferroptosis of neutrophils serves a critical role in the pathogenies of SLE

Models of invariant natural killer T cells derived from patient PBMCs also could show that the accumulation of Gb3 negatively affected their numbers and cell function [48], impacting even further on the immune system

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The analysis revealed the presence of two candidate NF-B binding sites in the promoter of EAAT3 (Fig