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29,30 However, in the periodontitis model, we observed a fundamental reprogramming of GSH metabolism in BM neutrophil, with IFN-I signaling markedly upregulating the degradation-regulatory protein Chac2, which can in turn repressed the catalytic activity of Chac1, 38,39,40,69 resulting in substantial intracellular GSH accumulation

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PMID: 39252968 iScienceHypoxia-induced HMGB1 promotes glioma stem cells self-renewal and tumorigenicity via RAGE

Single phospho-null or phospho-mimic mutants induce similar Golgi fragmentation levels to WT ArfGAP1 (85% cells)