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Therefore, targeting ABCG2 may be a promising therapy to eliminate LSCs

E-selectin, the expression of which on the surface of ECs, is induced by tumour necrosis factor (TNF) under an inflammatory stimulation, which is present at high levels in brain metastases, and preferentially contributes to shear-resistant adhesion and transendothelial migration by binding the transmembrane glycoprotein CD44 on BC cells to establish heterogenous adhesion

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A., Zhu C., Medvedeva V., et al., Improvement of Neuropathology and Transcriptional Deficits in CAG 140 Knockin Mice Supports a Beneficial Effect of Dietary Curcumin in Huntington's Disease, Molecular Neurodegeneration 7 (2012): 12