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Within osteomyelitis lesions, mitochondrial damage and metabolic stress can trigger multiple death signals simultaneously

Additional exposure of the organoids to P4 or MPA, together with activation of cAMP signaling, creates a decidualized endometrial phenotype comparable to the in vivo situation [53, 54, 64]

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Mitochondrial sorting into EVs involves active packaging mechanisms and may synergize with TNT-mediated transfer under certain conditions