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In MS lesions, TLR3 and 4 are expressed by microglia and astrocytes, where astroglial TLR expression is particularly prominent at later stages of inflammation, which may be instrumental in mitigating inflammation and promote tissue repair ( Furthermore, following acute inflammation and demyelination, hypertrophic astrocytes eventually form a glial scar in the center of white matter lesions ( Recent studies have implicated gut microbiota in immunological disorders including MS and its animal model, EAE (108, 109)

Noteworthy, these studies were carried out in non-neuronal cells

doi: 10.1007/s00262-018-2180-6

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