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doi: 10.1038/s42255-025-01396-6 62 LiY.InnocentinS.WithersD

Fibroblast proliferation leads to cardiac fibrosis, which replaces injured cardiomyocytes but excessive fibrosis leads to structural deformation, reduced heart function, ventricular enlargement, cardiomyocyte hypertrophy, increased wall stiffness, and progression to heart failure [7]