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Mild injection-site reactions affect approximately 5-10 percent of recipients, while systemic effects are less frequent but warrant careful monitoring

RIG-I, retinoic acid-inducible gene I

Likewise, inborn errors of metabolism that impair the upstream processing and trafficking of B 12 or folate lead to elevation of this metabolite, a condition collectively known as hyperhomocystinemia

Figure 2 Both insoluble fibrils and soluble A oligomers exert potent neurotoxicity by forming plaques and triggering pathological cascades, including synaptic failure, excitotoxicity, changes in membrane permeability, and chronic inflammation (77, 78)