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In a mouse model with I/R injury, the combination of cyclosporine A (CsA) and DFX synergistically inhibits I/R-induced iron overload, lipid peroxidation, and cardiomyocyte ferroptosis, reducing infarct size and improving poor cardiac remodeling after I/R injury

Such prevention released cGMP for continued activation of the NO/cGMP pathway and increased the blood flow of the penile tissues during sexual stimulation [3]