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found that endothelial cell dysfunction, which is characterized by low nitric oxide (NO) bioavailability, greater reactive oxygen species production, the activation of abscission enzymes, and impaired extracellular lysosome-associated organelle function, triggers the degradation of the EG (57), leading to shear stress defects

Further confirmation through Chip-qPCR demonstrated that, compared to the control group, the enrichment of H3K18la at the promoters of STEAP3 and HSD3B1 significantly decreased after DON treatment, while lactate supplementation ameliorated this effect (Fig

Topical retinoid, micro needling, radiofrequency, or fractional lasers, and combination therapy give the best improvements