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High levels of inflammation are present in virtually every chronic illness, like diabetes, heart disease, and cancer

Regarding the inflammatory component in neuronal disorders, ALA and DHLA inhibit inflammasomes in neural tissues by decreasing pro-inflammatory mediators, such as interleukin-2 (IL-2), IFN-, and TNF-, increasing anti-inflammatory cytokines such as interleukin-10 (IL-10), and regulating the transcription of nuclear factors Nrf2 and NF-B in physiological processes

Our analyses allowed us to establish a parallelism around the bi-directional interactions between host and bacteria, ultimately informing on the possible cause-effect relationship behind the observed phenotype

In a study done in 15 patients with PD, 200 mg of GSH was given intranasally and showed that GSH levels did not only raise, but the levels remained high even 1 hour after administration