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DDC may also be a substrate of phase II metabolism, which involves formation of diethyldithiomethylcarbamate (Me-DDC) and the glucuronic acid of DDC

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NAC cysteine compounds (in the reverse adjustment) play an important role in the synthesis of glutamate, with a portion provided by NAC cysteine by the sodium-dependent transport mechanism through the bloodbrain barrier, converted into cystine in the brain, and the latter provided via the cystine-glutamate transporter exchange to glutamate, which causes mGLuR2/3 receptor activation and leads to decreased synaptic glutamate release

Cell-cycle modification requires careful interpretation because its consequences may differ between healthy, senescent and malignant cells