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The hypothesis that B12 downregulates inflammatory genes of COVID-19 via methyl-dependent epigenetic mechanisms was proved by the hypermethylation of three CpGs located in the 5 UTR region and in the proximal portion of the first exon of CCL3 (Chr17: 36,090,276 - 36,090,005) of leukocytes from cultures of MOD and SEV groups treated with the vitamin

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The IGF-1 DES-induced concentration-response curves of EPSCs and fEPSPs were analyzed by one-way ANOVA and Newman-Keuls test for comparisons

The following case reports exemplify potential hybrid harms in Pfizer-vaccinated individuals who developed myocarditis after SARS-CoV-2 infection